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Telomere Shorting is how our bodies age-Lengthen Them & Live Longer

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Manage episode 235999435 series 1024467
Contenu fourni par AgingYounger. Tout le contenu du podcast, y compris les épisodes, les graphiques et les descriptions de podcast, est téléchargé et fourni directement par AgingYounger ou son partenaire de plateforme de podcast. Si vous pensez que quelqu'un utilise votre œuvre protégée sans votre autorisation, vous pouvez suivre le processus décrit ici https://fr.player.fm/legal.
Telomeres function similarly, preventing chromosomes from fraying or tangling with one another. When that happens, it can cause genetic information to get mixed up or destroyed, leading to cell malfunction, increasing the risk of disease or even shortening lifespan. Each time a cell divides, its telomeres become shorter. After years of splicing and dicing, telomeres become too short for more divisions. At this point, cells are unable to divide further and become inactive, or die. As more of our cells lose their telomeres and go out of commission, without others to take their place, the body follows and begins breaking down. Telomeres don’t leave (or shorten) quietly. Their shortening process has been linked with aging, cancer and a higher risk of death. A study found that individuals with the shortest telomeres, or about 10 percent of the study’s participants, were 23 percent more likely to die within three years than those with longer telomeres. There is an enzyme called Telomerase that lengthens telomeres and keeps them from wearing out too fast or too early, which helps you live longer. By reawakening telomerase in human cells where it’s stopped working, normal human aging could be slowed. Want to start Aging Younger? Tune in Wednesday JUNE 12TH at 7PM FOR THE REST OF THE STORY! http://tobtr.com/s/11375979
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301 episodes

Artwork
iconPartager
 
Manage episode 235999435 series 1024467
Contenu fourni par AgingYounger. Tout le contenu du podcast, y compris les épisodes, les graphiques et les descriptions de podcast, est téléchargé et fourni directement par AgingYounger ou son partenaire de plateforme de podcast. Si vous pensez que quelqu'un utilise votre œuvre protégée sans votre autorisation, vous pouvez suivre le processus décrit ici https://fr.player.fm/legal.
Telomeres function similarly, preventing chromosomes from fraying or tangling with one another. When that happens, it can cause genetic information to get mixed up or destroyed, leading to cell malfunction, increasing the risk of disease or even shortening lifespan. Each time a cell divides, its telomeres become shorter. After years of splicing and dicing, telomeres become too short for more divisions. At this point, cells are unable to divide further and become inactive, or die. As more of our cells lose their telomeres and go out of commission, without others to take their place, the body follows and begins breaking down. Telomeres don’t leave (or shorten) quietly. Their shortening process has been linked with aging, cancer and a higher risk of death. A study found that individuals with the shortest telomeres, or about 10 percent of the study’s participants, were 23 percent more likely to die within three years than those with longer telomeres. There is an enzyme called Telomerase that lengthens telomeres and keeps them from wearing out too fast or too early, which helps you live longer. By reawakening telomerase in human cells where it’s stopped working, normal human aging could be slowed. Want to start Aging Younger? Tune in Wednesday JUNE 12TH at 7PM FOR THE REST OF THE STORY! http://tobtr.com/s/11375979
  continue reading

301 episodes

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